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The Journal of General Virology|August 8, 2023
Open reading frames M12/M13 jointly contribute to MHV-68 latencyBeatrix Steer, Barbara Adler, Heiko Adler
Plos Pathogens|March 24, 2016
Multiple Lytic Origins of Replication Are Required for Optimal Gammaherpesvirus Fitness In Vitro and In VivoChristine Sattler, Beatrix Steer, Heiko Adler
Journal of Virology|April 20, 2007
Murine gammaherpesvirus 68 contains two functional lytic origins of replicationHeiko Adler, Beatrix Steer, Klaus Freimüller, et al.
Virology Journal|June 12, 2012
Murine gammaherpesvirus 68 glycoprotein 150 does not contribute to latency amplification in vivoRomana Ruiss, Shinji Ohno, Beatrix Steer, et al.
Trends in Microbiology|December 14, 2016
Herpesviruses and Their Host Cells: A Successful LiaisonBarbara Adler, Christine Sattler, Heiko Adler
European Journal of Immunology|November 30, 2018
B cells latently infected with murine gammaherpesvirus 68 (MHV-68) are present in the mouse thymus-A step toward immune evasion?Tomoyoshi Yamano, Madlen Steinert, Beatrix Steer, et al.
Plos One|August 22, 2007
A gammaherpesviral internal repeat contributes to latency amplificationNagendra N Thakur, Susanne El-Gogo, Beatrix Steer, et al.
Scientific Reports|August 27, 2016
The small noncoding RNAs (sncRNAs) of murine gammaherpesvirus 68 (MHV-68) are involved in regulating the latent-to-lytic switch in vivoBeatrix Steer, Martin Strehle, Christine Sattler, et al.
Cells|December 22, 2023
A Mouse Model to Study the Pathogenesis of γ-herpesviral Infections in Germinal Center B CellsUrsula Rambold, Stefanie Sperling, Zakir Chew, et al.
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