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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 6, 2012
ALS-associated ataxin 2 polyQ expansions enhance stress-induced caspase 3 activation and increase TDP-43 pathological modificationsMichael P Hart, Aaron D GitlerTrends in Genetics : TIG|April 29, 2022
Singling out motor neurons in the age of single-cell transcriptomicsJacob A Blum, Aaron D GitlerCell Cycle (Georgetown, Tex.)|April 16, 2003
Regulating heart development: the role of Nf1Aaron D Gitler, Jonathan A EpsteinJournal of Visualized Experiments : Jove|August 16, 2011
High-throughput yeast plasmid overexpression screenMichael S Fleming, Aaron D GitlerNeuron|January 6, 2017
Raise the Roof: Boosting the Efficacy of a Spinal Muscular Atrophy TherapyNicholas J Kramer, Aaron D GitlerCurrent Biology : CB|August 22, 2015
Neurodegeneration: A Leg Up on TDP-43Matthew D Figley, Aaron D GitlerNeuron|May 19, 2022
Why you always in a mood? Pumpin' polyP, actin' brand newKevin A Guttenplan, Aaron D GitlerJournal of Molecular Neuroscience : MN|June 11, 2011
Model organisms reveal insight into human neurodegenerative disease: ataxin-2 intermediate-length polyglutamine expansions are a risk factor for ALSNancy M Bonini, Aaron D GitlerPageof 189