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International Journal of Cancer
|
November 26, 2003
p53 polymorphic variants at codon 72 exert different effects on cell cycle progression
David Pim, Lawrence Banks
APMIS : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica
|
June 18, 2010
Interaction of viral oncoproteins with cellular target molecules: infection with high-risk vs low-risk human papillomaviruses
David Pim, Lawrence Banks
Oncogene
|
November 22, 2002
Chimaeric HPV E6 proteins allow dissection of the proteolytic pathways regulating different E6 cellular target proteins
David Pim, Miranda Thomas, Lawrence Banks
Trends in Biochemical Sciences
|
August 23, 2003
Viruses and the 26S proteasome: hacking into destruction
Lawrence Banks, David Pim, Miranda Thomas
International Journal of Cancer
|
June 2, 2004
HPV-18 E6*I modulates HPV-18 full-length E6 functions in a cell cycle dependent manner
Ernesto Guccione, David Pim, Lawrence Banks
Journal of Virology
|
July 31, 2009
The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 protein
David Pim, Vjekoslav Tomaic, Lawrence Banks
Nature Reviews. Cancer
|
November 24, 2012
Human tumour viruses and the deregulation of cell polarity in cancer
Lawrence Banks, David Pim, Miranda Thomas
Virology
|
August 25, 2009
The stability of the human papillomavirus E6 oncoprotein is E6AP dependent
Vjekoslav Tomaić, David Pim, Lawrence Banks
Current Opinion in Cell Biology
|
May 26, 2019
Choosing the right path: membrane trafficking and infectious entry of small DNA tumor viruses
Marina Bugnon Valdano, David Pim, Lawrence Banks
Frontiers in Cellular and Infection Microbiology
|
June 14, 2019
Diverse Papillomavirus Types Induce Endosomal Tubulation
Abida Siddiqa, Paola Massimi, David Pim, et al.
Page
of 4
Search research articles
Search
Showing results (1-10 of 32) with videos related to
Sort By:
Page
of 4
International Journal of Cancer
|
November 26, 2003
p53 polymorphic variants at codon 72 exert different effects on cell cycle progression
David Pim, Lawrence Banks
APMIS : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica
|
June 18, 2010
Interaction of viral oncoproteins with cellular target molecules: infection with high-risk vs low-risk human papillomaviruses
David Pim, Lawrence Banks
Oncogene
|
November 22, 2002
Chimaeric HPV E6 proteins allow dissection of the proteolytic pathways regulating different E6 cellular target proteins
David Pim, Miranda Thomas, Lawrence Banks
Trends in Biochemical Sciences
|
August 23, 2003
Viruses and the 26S proteasome: hacking into destruction
Lawrence Banks, David Pim, Miranda Thomas
International Journal of Cancer
|
June 2, 2004
HPV-18 E6*I modulates HPV-18 full-length E6 functions in a cell cycle dependent manner
Ernesto Guccione, David Pim, Lawrence Banks
Journal of Virology
|
July 31, 2009
The human papillomavirus (HPV) E6* proteins from high-risk, mucosal HPVs can direct degradation of cellular proteins in the absence of full-length E6 protein
David Pim, Vjekoslav Tomaic, Lawrence Banks
Nature Reviews. Cancer
|
November 24, 2012
Human tumour viruses and the deregulation of cell polarity in cancer
Lawrence Banks, David Pim, Miranda Thomas
Virology
|
August 25, 2009
The stability of the human papillomavirus E6 oncoprotein is E6AP dependent
Vjekoslav Tomaić, David Pim, Lawrence Banks
Current Opinion in Cell Biology
|
May 26, 2019
Choosing the right path: membrane trafficking and infectious entry of small DNA tumor viruses
Marina Bugnon Valdano, David Pim, Lawrence Banks
Frontiers in Cellular and Infection Microbiology
|
June 14, 2019
Diverse Papillomavirus Types Induce Endosomal Tubulation
Abida Siddiqa, Paola Massimi, David Pim, et al.
Page
of 4