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Diabetes|January 1, 1986
Islet allografts are destroyed by disease occurrence in the spontaneously diabetic BB ratS J Prowse, D Bellgrau, K J LaffertyHormone and Metabolic Research = Hormon- Und Stoffwechselforschung = Hormones Et Metabolisme|June 1, 1996
The diabetic BB rat. Neither Th1 nor Th2?D Bellgrau, D Stenger, C Richards, et al.Diabetes|January 1, 1994
Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB ratD Bellgrau, J M Redd, K S SellinsJournal of Virology|May 1, 1988
Recognition of adenovirus E1A gene products on immortalized cell surfaces by cytotoxic T lymphocytesD Bellgrau, T A Walker, J L CookDiabetologia|January 1, 1996
Resistance to tolerance induction in the diabetes-prone biobreeding rat as one manifestation of abnormal responses to superantigensK S Sellins, D P Gold, D BellgrauScience (New York, N.Y.)|September 18, 1981
Spontaneous diabetes in rats: destruction of islets is prevented by immunological toleranceA Naji, W K Silvers, D Bellgrau, et al.Transplantation|August 1, 1990
Cyclosporine-induced autoimmunity in rats carrying thymus allograftsS K Babcock, K Niswender, D B Wilson, et al.Cancer Immunology, Immunotherapy : CII|January 1, 1992
Allogeneic tumor-specific cytotoxic T lymphocytesJ M Redd, A C Lagarde, C A Kruse, et al.European Journal of Immunology|July 1, 1988
A requirement for the CD5 antigen in T cell activationM J McAteer, A C Lagarde, H M Georgiou, et al.Oncogene|August 15, 1996
E1A oncogene expression in target cells induces cytolytic susceptibility at a post-recognition stage in the interaction with killer lymphocytesJ L Cook, T A Potter, D Bellgrau, et al.Pageof 190