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The Journal of Cell Biology|May 14, 2003
Amyloid as a natural productJeffery W Kelly, William E BalchNature Chemical Biology|April 19, 2006
The integration of cell and chemical biology in protein foldingJeffery W Kelly, William E BalchFASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|August 9, 2012
Translational attenuation differentially alters the fate of disease-associated fibulin proteinsJohn D Hulleman, William E Balch, Jeffery W KellyCurrent Opinion in Structural Biology|December 17, 2003
Pathological and functional amyloid formation orchestrated by the secretory pathwayMary E Huff, William E Balch, Jeffery W KellyMolecular Biology of the Cell|October 28, 2011
Compromised mutant EFEMP1 secretion associated with macular dystrophy remedied by proteostasis network alterationJohn D Hulleman, Shalesh Kaushal, William E Balch, et al.Science (New York, N.Y.)|February 16, 2008
Adapting proteostasis for disease interventionWilliam E Balch, Richard I Morimoto, Andrew Dillin, et al.Critical Reviews in Biochemistry and Molecular Biology|February 25, 2012
Gelsolin amyloidosis: genetics, biochemistry, pathology and possible strategies for therapeutic interventionJames P Solomon, Lesley J Page, William E Balch, et al.Journal of Molecular Biology|November 5, 2003
Gelsolin domain 2 Ca2+ affinity determines susceptibility to furin proteolysis and familial amyloidosis of finnish typeMary E Huff, Lesley J Page, William E Balch, et al.Biochemical and Biophysical Research Communications|September 1, 2004
Ca2+ binding protects against gelsolin amyloidosisLesley J Page, Mary E Huff, Jeffery W Kelly, et al.Trends in Biochemical Sciences|April 7, 2007
Functional amyloid--from bacteria to humansDouglas M Fowler, Atanas V Koulov, William E Balch, et al.Pageof 40