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American Journal of Physiology. Renal Physiology
|
June 6, 2014
APOL1 risk variants enhance podocyte necrosis through compromising lysosomal membrane permeability
Xiqian Lan, Aakash Jhaveri, Kang Cheng, et al.
Journal of the American Society of Nephrology : JASN
|
December 3, 2011
APOL1 risk variants predict histopathology and progression to ESRD in HIV-related kidney disease
Derek M Fine, Walter G Wasser, Michelle M Estrella, et al.
Pediatric Nephrology (Berlin, Germany)
|
August 8, 2020
Kidney failure risk in type 1 vs. type 2 childhood-onset diabetes mellitus
Oren Pleniceanu, Gilad Twig, Dorit Tzur, et al.
The Journal of Biological Chemistry
|
February 9, 2010
Kidney-specific overexpression of Sirt1 protects against acute kidney injury by retaining peroxisome function
Kazuhiro Hasegawa, Shu Wakino, Kyoko Yoshioka, et al.
Scientific Reports
|
March 7, 2019
Disruption of APOL1-miR193a Axis Induces Disorganization of Podocyte Actin Cytoskeleton
Vinod Kumar, Nitpriya Paliwal, Kamesh Ayasolla, et al.
Biomolecules
|
January 8, 2025
APOL1 Modulates Renin-Angiotensin System
Vinod Kumar, Prabhjot Kaur, Kameshwar Ayasolla, et al.
Human Molecular Genetics
|
February 11, 2010
African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans
Doron M Behar, Saharon Rosset, Shay Tzur, et al.
Blood Cells, Molecules & Diseases
|
October 14, 2015
A founder effect for p47(phox)Trp193Ter chronic granulomatous disease in Kavkazi Jews
Martin de Boer, Shay Tzur, Karin van Leeuwen, et al.
The Journal of Investigative Dermatology
|
December 21, 2007
Normophosphatemic familial tumoral calcinosis is caused by deleterious mutations in SAMD9, encoding a TNF-alpha responsive protein
Ilana Chefetz, Danny Ben Amitai, Sarah Browning, et al.
Iscience
|
January 24, 2022
Endoplasmic reticulum-translocation is essential for APOL1 cellular toxicity
Etty Kruzel-Davila, Ira Bavli-Kertselli, Ayala Ofir, et al.
Page
of 14
Search research articles
Search
Showing results (91-100 of 134) with videos related to
Sort By:
Page
of 14
American Journal of Physiology. Renal Physiology
|
June 6, 2014
APOL1 risk variants enhance podocyte necrosis through compromising lysosomal membrane permeability
Xiqian Lan, Aakash Jhaveri, Kang Cheng, et al.
Journal of the American Society of Nephrology : JASN
|
December 3, 2011
APOL1 risk variants predict histopathology and progression to ESRD in HIV-related kidney disease
Derek M Fine, Walter G Wasser, Michelle M Estrella, et al.
Pediatric Nephrology (Berlin, Germany)
|
August 8, 2020
Kidney failure risk in type 1 vs. type 2 childhood-onset diabetes mellitus
Oren Pleniceanu, Gilad Twig, Dorit Tzur, et al.
The Journal of Biological Chemistry
|
February 9, 2010
Kidney-specific overexpression of Sirt1 protects against acute kidney injury by retaining peroxisome function
Kazuhiro Hasegawa, Shu Wakino, Kyoko Yoshioka, et al.
Scientific Reports
|
March 7, 2019
Disruption of APOL1-miR193a Axis Induces Disorganization of Podocyte Actin Cytoskeleton
Vinod Kumar, Nitpriya Paliwal, Kamesh Ayasolla, et al.
Biomolecules
|
January 8, 2025
APOL1 Modulates Renin-Angiotensin System
Vinod Kumar, Prabhjot Kaur, Kameshwar Ayasolla, et al.
Human Molecular Genetics
|
February 11, 2010
African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans
Doron M Behar, Saharon Rosset, Shay Tzur, et al.
Blood Cells, Molecules & Diseases
|
October 14, 2015
A founder effect for p47(phox)Trp193Ter chronic granulomatous disease in Kavkazi Jews
Martin de Boer, Shay Tzur, Karin van Leeuwen, et al.
The Journal of Investigative Dermatology
|
December 21, 2007
Normophosphatemic familial tumoral calcinosis is caused by deleterious mutations in SAMD9, encoding a TNF-alpha responsive protein
Ilana Chefetz, Danny Ben Amitai, Sarah Browning, et al.
Iscience
|
January 24, 2022
Endoplasmic reticulum-translocation is essential for APOL1 cellular toxicity
Etty Kruzel-Davila, Ira Bavli-Kertselli, Ayala Ofir, et al.
Page
of 14