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Dementia and Geriatric Cognitive Disorders|November 24, 1999
No detected mutations in the genes for the amyloid precursor protein and presenilins 1 and 2 in a swiss early-onset Alzheimer's disease family with a dominant mode of inheritanceA Savioz, G Leuba, C Forsell, et al.
Scandinavian Journal of Clinical and Laboratory Investigation|November 1, 1993
Genetic carrier detection in Norwegian families with acute intermittent porphyriaE Sagen, A Laegreid, M Anvret, et al.
Multiple Sclerosis (Houndmills, Basingstoke, England)|May 7, 2002
APOE genotypes and disease severity in multiple sclerosisT Masterman, Z Zhang, D Hellgren, et al.
Acta Neurologica Scandinavica|February 1, 1995
Microsatellite D21S210 (GT-12) allele frequencies in sporadic Alzheimer's diseaseL Lannfelt, L Lilius, M Viitanen, et al.
Neuroreport|April 17, 2001
Increased risk for frontotemporal dementia through interaction between tau polymorphisms and apolipoprotein E epsilon4M Ingelson, S F Fabre, L Lilius, et al.
Neuroscience Letters|February 28, 1994
Amyloid precursor protein mutation causes Alzheimer's disease in a Swedish familyL Lannfelt, N Bogdanovic, H Appelgren, et al.
Neurology|May 5, 1999
A DLST genotype associated with reduced risk for Alzheimer's diseaseK F Sheu, A M Brown, B S Kristal, et al.
Molecular Psychiatry|November 17, 2005
Genome scan on Swedish Alzheimer's disease familiesA Sillén, C Forsell, L Lilius, et al.
Neuroscience Letters|March 14, 1994
Lack of association between apolipoprotein E allele epsilon 4 and sporadic Alzheimer's diseaseL Lannfelt, L Lilius, M Nastase, et al.
Neuroscience Letters|January 22, 2000
Tau gene polymorphisms and apolipoprotein E epsilon4 may interact to increase risk for Alzheimer's diseaseL Lilius, S Froelich Fabre, H Basun, et al.
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