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Journal of Cellular Physiology|August 26, 2011
Constitutively active PTH/PTHrP receptor specifically expressed in osteoblasts enhances bone formation induced by bone marrow ablationNoriaki Ono, Kazuhisa Nakashima, Ernestina Schipani, et al.JBMR Plus|July 27, 2019
Profilin 1 Negatively Regulates Osteoclast Migration in Postnatal Skeletal Growth, Remodeling, and Homeostasis in MiceJumpei Shirakawa, Shuhei Kajikawa, Ralph T Böttcher, et al.Journal of Bone and Mineral Metabolism|May 17, 2014
Hindlimb-unloading suppresses B cell population in the bone marrow and peripheral circulation associated with OPN expression in circulating blood cellsYoichi Ezura, Junji Nagata, Masashi Nagao, et al.Frontiers in Cell and Developmental Biology|July 11, 2024
Patient-derived and gene-edited pluripotent stem cells lacking <i>NPHP1</i> recapitulate juvenile nephronophthisis in abnormalities of primary cilia and renal cyst formationYutaka Arai, Hidenori Ito, Tomoya Shimizu, et al.Journal of Cellular Physiology|January 3, 2013
Nck1 deficiency accelerates unloading-induced bone lossA C Smriti Aryal, Kentaro Miyai, Tadayoshi Hayata, et al.The Journal of Biological Chemistry|November 14, 2008
Angiotensin II type 2 receptor blockade increases bone massYayoi Izu, Fumitaka Mizoguchi, Aya Kawamata, et al.Journal of Cellular Physiology|September 27, 2007
OPN deficiency suppresses appearance of odontoclastic cells and resorption of the tooth root induced by experimental force applicationChooryung J Chung, Kunimichi Soma, Susan R Rittling, et al.JBMR Plus|September 13, 2023
Identification of a Biallelic Missense Variant in Gasdermin D (c.823G > C, p.Asp275His) in a Patient of Atypical Gorham-Stout Disease in a Consanguineous FamilyDaniela Tiaki Uehara, Tomoki Muramatsu, Senichi Ishii, et al.Journal of Cellular Biochemistry|February 2, 2008
JunD suppresses bone formation and contributes to low bone mass induced by estrogen depletionAya Kawamata, Yayoi Izu, Haruna Yokoyama, et al.Biochemical and Biophysical Research Communications|March 18, 2018
Dok-3 and Dok-1/-2 adaptors play distinctive roles in cell fusion and proliferation during osteoclastogenesis and cooperatively protect mice from osteopeniaShuhei Kajikawa, Yuu Taguchi, Tadayoshi Hayata, et al.Pageof 7