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Autoimmunity Reviews|August 8, 2006
Dendritic cell subsets and type I diabetes: focus upon DC-based therapyJeannette Lo, Michael J Clare-SalzlerBiomaterials|May 30, 2009
A high-throughput microparticle microarray platform for dendritic cell-targeting vaccinesAbhinav P Acharya, Michael J Clare-Salzler, Benjamin G KeselowskyCurrent Opinion in Organ Transplantation|May 16, 2009
Extracorporeal photopheresis-induced immune tolerance: a focus on modulation of antigen-presenting cells and induction of regulatory T cells by apoptotic cellsChang-Qing Xia, Kim A Campbell, Michael J Clare-SalzlerBiochemical and Biophysical Research Communications|December 13, 2006
Dendritic cells post-maturation are reprogrammed with heightened IFN-gamma and IL-10Chang-Qing Xia, Ruihua Peng, Mani Annamalai, et al.Journal of Immunology Research|April 14, 2018
Immature Dendritic Cell Therapy Confers Durable Immune Modulation in an Antigen-Dependent and Antigen-Independent Manner in Nonobese Diabetic MiceJeannette Lo, Chang-Qing Xia, Ruihua Peng, et al.Thyroid : Official Journal of the American Thyroid Association|December 31, 2010
Parvovirus B19 infection in Hashimoto's thyroiditis, papillary thyroid carcinoma, and anaplastic thyroid carcinomaLaura A Adamson, Larry J Fowler, Michael J Clare-Salzler, et al.Biomaterials|October 3, 2008
Adhesive substrate-modulation of adaptive immune responsesAbhinav P Acharya, Natalia V Dolgova, Michael J Clare-Salzler, et al.Journal of Blood Disorders & Transfusion|January 19, 2016
Characterization of Bone Marrow-Derived Dendritic Cells Developed in Serum-Free Media and their Ability to Prevent Type 1 Diabetes in Nonobese Diabetic MiceBen M Looney, Anna V Chernatynskaya, Michael J Clare-Salzler, et al.Annals of the New York Academy of Sciences|November 30, 2006
Upregulation of Foxp3 expression in mouse and human Treg is IL-2/STAT5 dependent: implications for the NOD STAT5B mutation in diabetes pathogenesisMatthew R Murawski, Sally A Litherland, Michael J Clare-Salzler, et al.Annals of the New York Academy of Sciences|November 30, 2006
Heightened interferon-alpha/beta response causes myeloid cell dysfunction and promotes T1D pathogenesis in NOD miceRui Hua Peng, Edward Paek, Chanqing Q Xia, et al.Pageof 6