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Oncotarget|September 28, 2014
AP4 is required for mitogen- and c-MYC-induced cell cycle progressionRene Jackstadt, Heiko HermekingBiochimica Et Biophysica Acta|April 15, 2014
MicroRNAs as regulators and mediators of c-MYC functionRene Jackstadt, Heiko HermekingMethods in Molecular Biology (Clifton, N.J.)|September 6, 2013
Genome-wide analysis of c-MYC-regulated mRNAs and miRNAs, and c-MYC DNA binding by next-generation sequencingRene Jackstadt, Antje Menssen, Heiko HermekingMethods in Molecular Biology (Clifton, N.J.)|May 21, 2021
Genome-Wide Analysis of c-MYC-Regulated mRNAs and miRNAs and c-MYC DNA-Binding by Next-Generation SequencingRene Jackstadt, Markus Kaller, Antje Menssen, et al.Oncotarget|September 7, 2013
Repression of c-Kit by p53 is mediated by miR-34 and is associated with reduced chemoresistance, migration and stemnessHelge Siemens, Rene Jackstadt, Markus Kaller, et al.The EMBO Journal|November 5, 2013
SNAIL and miR-34a feed-forward regulation of ZNF281/ZBP99 promotes epithelial-mesenchymal transitionStefanie Hahn, Rene Jackstadt, Helge Siemens, et al.Cancer Research|November 29, 2013
p53-induced miR-15a/16-1 and AP4 form a double-negative feedback loop to regulate epithelial-mesenchymal transition and metastasis in colorectal cancerLei Shi, Rene Jackstadt, Helge Siemens, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|December 18, 2012
Detection of miR-34a promoter methylation in combination with elevated expression of c-Met and β-catenin predicts distant metastasis of colon cancerHelge Siemens, Jens Neumann, Rene Jackstadt, et al.Cell Cycle (Georgetown, Tex.)|December 3, 2011
miR-34 and SNAIL form a double-negative feedback loop to regulate epithelial-mesenchymal transitionsHelge Siemens, Rene Jackstadt, Sabine Hünten, et al.Plos One|August 12, 2014
Colorectal cancers mimic structural organization of normal colonic cryptsLaura Cernat, Cristina Blaj, Rene Jackstadt, et al.Pageof 15