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Mbio|August 10, 2017
Bacterial-Chromatin Structural Proteins Regulate the Bimodal Expression of the Locus of Enterocyte Effacement (LEE) Pathogenicity Island in Enteropathogenic Escherichia coliHervé Leh, Ahmad Khodr, Marie-Christine Bouger, et al.
Infection and Immunity|April 29, 2015
H-NS Silencing of the Salmonella Pathogenicity Island 6-Encoded Type VI Secretion System Limits Salmonella enterica Serovar Typhimurium Interbacterial KillingYannick R Brunet, Ahmad Khodr, Laureen Logger, et al.
Journal of Molecular Biology|November 11, 2003
Crystal structure of the N-terminal dimerisation domain of VicH, the H-NS-like protein of Vibrio choleraeRachel Cerdan, Vanessa Bloch, Yinshan Yang, et al.
Nature Structural Biology|February 20, 2003
The H-NS dimerization domain defines a new fold contributing to DNA recognitionVanessa Bloch, Yinshan Yang, Emmanuel Margeat, et al.
Biochimica Et Biophysica Acta. Bioenergetics|July 6, 2024
Functional organization of 3D plant thylakoid membranes as seen by high resolution microscopySimona Streckaite, Cristian Ilioaia, Igor Chaussavoine, et al.
Plos Pathogens|November 15, 2019
The Crohn's disease-associated Escherichia coli strain LF82 relies on SOS and stringent responses to survive, multiply and tolerate antibiotics within macrophagesGaëlle Demarre, Victoria Prudent, Hanna Schenk, et al.
Journal of the American Chemical Society|January 23, 2010
Measurement of long-range interatomic distances by solid-state tritium-NMR spectroscopyAlexander K L Yuen, Olivier Lafon, Thibault Charpentier, et al.
Nucleic Acids Research|September 21, 2007
High-affinity DNA binding sites for H-NS provide a molecular basis for selective silencing within proteobacterial genomesBenjamin Lang, Nicolas Blot, Emeline Bouffartigues, et al.
Communications Biology|May 26, 2021
The Crohn's disease-related bacterial strain LF82 assembles biofilm-like communities to protect itself from phagolysosomal attackVictoria Prudent, Gaëlle Demarre, Emilie Vazeille, et al.
Cancer Discovery|July 24, 2021
Actinomycin D Targets NPM1c-Primed Mitochondria to Restore PML-Driven Senescence in AML TherapyHsin-Chieh Wu, Domitille Rérolle, Caroline Berthier, et al.
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