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T Deak

Showing results (31-40 of 61) with videos related to

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Endocrinology|December 1, 1995
A long-term increase in basal levels of corticosterone and a decrease in corticosteroid-binding globulin after acute stressor exposureM Fleshner, T Deak, R L Spencer, et al.
British Journal of Pharmacology|March 23, 2013
N-Arachidonoyl glycine suppresses Na⁺/Ca²⁺ exchanger-mediated Ca²⁺ entry into endothelial cells and activates BK(Ca) channels independently of GPCRsAlexander I Bondarenko, Konstantin Drachuk, Olga Panasiuk, et al.
Journal of Cleaner Production|May 6, 2026
Designing cost-effective supply chains for plastics at the end-of-lifeBaibhaw Kumar, Jean Pimentel, Natalia A Cano-Londoño, et al.
Methods in Molecular Biology (Clifton, N.J.)|September 12, 2018
High-Resolution Imaging of STIM/Orai Subcellular Localization Using Array Confocal Laser Scanning MicroscopyAndras T Deak, Benjamin Gottschalk, Emrah Eroglu, et al.
Brain Research Bulletin|January 1, 1997
TNF-alpha-induced corticosterone elevation but not serum protein or corticosteroid binding globulin reduction is vagally mediatedM Fleshner, L Silbert, T Deak, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 18, 1998
Exposure to acute stress induces brain interleukin-1beta protein in the ratK T Nguyen, T Deak, S M Owens, et al.
Journal of Neuroendocrinology|April 15, 2014
Novel aspects of glucocorticoid actionsE T Uchoa, G Aguilera, J P Herman, et al.
Brain Research|November 14, 1998
The long term acute phase-like responses that follow acute stressor exposure are blocked by alpha-melanocyte stimulating hormoneE D Milligan, K T Nguyen, T Deak, et al.
Brain Research|November 27, 1999
Long-term changes in mineralocorticoid and glucocorticoid receptor occupancy following exposure to an acute stressorT Deak, K T Nguyen, C S Cotter, et al.
Journal of Cell Science|December 15, 2012
The endocannabinoid N-arachidonoyl glycine (NAGly) inhibits store-operated Ca2+ entry by preventing STIM1-Orai1 interactionAndras T Deak, Lukas N Groschner, Muhammad Rizwan Alam, et al.
Pageof 7

Showing results (31-40 of 61) with videos related to

Sort By:
Pageof 7
Endocrinology|December 1, 1995
A long-term increase in basal levels of corticosterone and a decrease in corticosteroid-binding globulin after acute stressor exposureM Fleshner, T Deak, R L Spencer, et al.
British Journal of Pharmacology|March 23, 2013
N-Arachidonoyl glycine suppresses Na⁺/Ca²⁺ exchanger-mediated Ca²⁺ entry into endothelial cells and activates BK(Ca) channels independently of GPCRsAlexander I Bondarenko, Konstantin Drachuk, Olga Panasiuk, et al.
Journal of Cleaner Production|May 6, 2026
Designing cost-effective supply chains for plastics at the end-of-lifeBaibhaw Kumar, Jean Pimentel, Natalia A Cano-Londoño, et al.
Methods in Molecular Biology (Clifton, N.J.)|September 12, 2018
High-Resolution Imaging of STIM/Orai Subcellular Localization Using Array Confocal Laser Scanning MicroscopyAndras T Deak, Benjamin Gottschalk, Emrah Eroglu, et al.
Brain Research Bulletin|January 1, 1997
TNF-alpha-induced corticosterone elevation but not serum protein or corticosteroid binding globulin reduction is vagally mediatedM Fleshner, L Silbert, T Deak, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 18, 1998
Exposure to acute stress induces brain interleukin-1beta protein in the ratK T Nguyen, T Deak, S M Owens, et al.
Journal of Neuroendocrinology|April 15, 2014
Novel aspects of glucocorticoid actionsE T Uchoa, G Aguilera, J P Herman, et al.
Brain Research|November 14, 1998
The long term acute phase-like responses that follow acute stressor exposure are blocked by alpha-melanocyte stimulating hormoneE D Milligan, K T Nguyen, T Deak, et al.
Brain Research|November 27, 1999
Long-term changes in mineralocorticoid and glucocorticoid receptor occupancy following exposure to an acute stressorT Deak, K T Nguyen, C S Cotter, et al.
Journal of Cell Science|December 15, 2012
The endocannabinoid N-arachidonoyl glycine (NAGly) inhibits store-operated Ca2+ entry by preventing STIM1-Orai1 interactionAndras T Deak, Lukas N Groschner, Muhammad Rizwan Alam, et al.
Pageof 7