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Diabetes|December 25, 2003
IL-1 receptor deficiency slows progression to diabetes in the NOD mouseHelen E Thomas, Windy Irawaty, Rima Darwiche, et al.Immunity|June 11, 2004
NKG2D blockade prevents autoimmune diabetes in NOD miceKouetsu Ogasawara, Jessica A Hamerman, Lauren R Ehrlich, et al.Plos One|December 2, 2015
E2-2 Dependent Plasmacytoid Dendritic Cells Control Autoimmune DiabetesLisbeth Hansen, Anja Schmidt-Christensen, Shashank Gupta, et al.Immunity|October 13, 2009
T cell islet accumulation in type 1 diabetes is a tightly regulated, cell-autonomous eventGreig P Lennon, Maria Bettini, Amanda R Burton, et al.Proceedings of the National Academy of Sciences of the United States of America|February 13, 2013
Antidiabetogenic MHC class II promotes the differentiation of MHC-promiscuous autoreactive T cells into FOXP3+ regulatory T cellsSue Tsai, Pau Serra, Xavier Clemente-Casares, et al.Cellular & Molecular Immunology|March 27, 2023
A T follicular helper cell origin for T regulatory type 1 cellsPatricia Solé, Jun Yamanouchi, Josep Garnica, et al.Nature Communications|June 7, 2022
Re-programming mouse liver-resident invariant natural killer T cells for suppressing hepatic and diabetogenic autoimmunityChannakeshava Sokke Umeshappa, Patricia Solé, Jun Yamanouchi, et al.The American Journal of Pathology|June 7, 2011
Autoreactive cytotoxic T lymphocytes acquire higher expression of cytotoxic effector markers in the islets of NOD mice after priming in pancreatic lymph nodesKate L Graham, Balasubramanian Krishnamurthy, Stacey Fynch, et al.The Review of Diabetic Studies : RDS|November 10, 2010
Turning human epidermis into pancreatic endodermPere Santamaria, Ignacio Rodriguez-Piza, Xavier Clemente-Casares, et al.Journal of Immunology (Baltimore, Md. : 1950)|August 29, 2014
The cross-priming capacity and direct presentation potential of an autoantigen are separable and inversely related propertiesJinguo Wang, Roopa Hebbandi Nanjundappa, Afshin Shameli, et al.Pageof 1,638