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Seminars in Immunopathology|February 7, 2013
Neutrophil proteinase 3 and dipeptidyl peptidase I (cathepsin C) as pharmacological targets in granulomatosis with polyangiitis (Wegener granulomatosis)Brice Korkmaz, Adam Lesner, Stephanie Letast, et al.Seminars in Immunopathology|April 5, 2013
The unfolded protein response triggered by environmental factorsMasanori KitamuraSeminars in Immunopathology|April 19, 2013
Neutrophil extracellular chromatin traps connect innate immune response to autoimmunityMarko Radic, Tony N MarionSeminars in Immunopathology|January 30, 2010
Th17 cytokines and vaccine-induced immunityYinyao Lin, Samantha R Slight, Shabaana A KhaderSeminars in Immunopathology|February 2, 2010
Costimulation of Th17 cells: Adding fuel or putting out the fire in the inflamed gut?Zili Zhang, James T Rosenbaum, Wenwei Zhong, et al.Seminars in Immunopathology|November 4, 2017
Newborn susceptibility to infection vs. disease depends on complex in vivo interactions of host and pathogenByron Brook, Danny Harbeson, Rym Ben-Othman, et al.Seminars in Immunopathology|October 18, 2017
The maternal microbiome during pregnancy and allergic disease in the offspringPeter J Vuillermin, Laurence Macia, Ralph Nanan, et al.Seminars in Immunopathology|November 24, 2017
The properdin pathway: an "alternative activation pathway" or a "critical amplification loop" for C3 and C5 activation?Richard A HarrisonSeminars in Immunopathology|November 25, 2017
Early life origin of type 1 diabetesMikael Knip, Kristiina Luopajärvi, Taina HärkönenPageof 91