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Both Pgp and MRP1 activities using calcein-AM contribute to drug resistance in AML

O Legrand1, G Simonin, J Y Perrot

  • 1EA1529, Université Paris VI, Formation de Recherche Claude Bernard, France.

Insights

Functional testing using calcein-AM uptake and efflux accurately measures P-glycoprotein (Pgp) and multidrug resistance-associated protein 1 (MRP1) functions in acute myeloid leukemia (AML). These functional assays, along with modulators like probenecid and CsA, show prognostic value for predicting complete remission (CR) in AML patients.

Area of Science:

  • Pharmacology and Toxicology
  • Cancer Biology
  • Hematology

Background:

  • P-glycoprotein (Pgp) and multidrug resistance-associated protein 1 (MRP1) are key efflux transporters implicated in multidrug resistance (MDR) in cancer.
  • Accurate functional assessment of Pgp and MRP1 is crucial for understanding drug resistance mechanisms and developing effective therapeutic strategies in acute myeloid leukemia (AML).
  • Existing methods for assessing transporter function may not always correlate directly with their clinical impact.

Purpose of the Study:

  • To evaluate the utility of calcein-AM uptake and calcein efflux assays for measuring Pgp and MRP1 functions, respectively.
  • To correlate these functional assessments with Pgp and MRP1 expression levels in cell lines and primary AML samples.
  • To determine the prognostic value of Pgp and MRP1 activity in predicting treatment outcomes, specifically complete remission (CR), in AML patients.

Main Methods:

  • Utilized thirteen cell lines with varying Pgp and MRP1 expression levels to validate calcein-AM uptake (for Pgp) and calcein efflux (for MRP1) assays.
  • Assessed the modulatory effects of cyclosporine A (CsA) on calcein-AM uptake and probenecid on calcein efflux to confirm transporter function.
  • Analyzed fresh leukemia cells from 53 AML patients, measuring Pgp and MRP1 expression (RT/PCR, flow cytometry) and function (calcein assays with modulators).

Main Results:

  • High correlations were observed between Pgp expression and CsA-modulated calcein-AM uptake (r = 0.96, p < 0.0001) and between MRP1 expression and probenecid-modulated calcein efflux (r = 0.91, p = 0.0003) in cell lines.
  • Similar strong correlations were found in AML patients: Pgp expression correlated with CsA effect on calcein-AM uptake (r = 0.83, p < 0.0001), and MRP1 expression correlated with probenecid effect on calcein fluorescence (r = 0.92, p < 0.0001).
  • Pgp activity was significantly higher in CD34+ AML cells, while MRP1 activity was higher in CD34- AML cells. Pgp expression/activity and MRP1 activity were identified as significant prognostic factors for achieving CR.

Conclusions:

  • Calcein-AM uptake and efflux assays, in conjunction with modulators like CsA and probenecid, provide reliable functional measurements of Pgp and MRP1 activity.
  • Functional assessment of Pgp and MRP1 exhibits significant prognostic value for predicting complete remission in acute myeloid leukemia patients.
  • MRP1 plays a contributing role in mediating drug resistance within the AML context, highlighting its importance as a therapeutic target.

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