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Related Concept Videos

Visual Agnosia01:12

Visual Agnosia

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Visual agnosia is a condition characterized by the inability to recognize visually presented objects despite having normal vision. For instance, a person with visual agnosia can describe the shape and color of an object but cannot identify or name it. This impairment does not affect their visual field, acuity, color vision, brightness discrimination, language, or memory. An example of this condition in a social setting is someone at a dinner party asking for "that silver thing with a round...
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Photoreceptors and Visual Pathways01:22

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At the molecular level, visual signals trigger transformations in photopigment molecules, resulting in changes in the photoreceptor cell's membrane potential. The photon's energy level is denoted by its wavelength, with each specific wavelength of visible light associated with a distinct color. The spectral range of visible light, classified as electromagnetic radiation, spans from 380 to 720 nm. Electromagnetic radiation wavelengths exceeding 720 nm fall under the infrared category,...
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Prosopagnosia01:24

Prosopagnosia

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Prosopagnosia, also known as face blindness, is the inability to recognize faces. In severe cases, individuals with prosopagnosia may not recognize close family members, including parents and spouses, by their faces. For instance, someone with prosopagnosia might walk past their child in a crowd, only realizing their mistake upon noticing their child's distinctive backpack or favorite jacket. Prosopagnosia specifically impairs facial recognition, while the recognition of other objects or...
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Visual System01:26

Visual System

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Light enters the eye through the cornea, a transparent, dome-shaped surface covering the surface of the eyeball that helps to direct and focus incoming light. This light is then channeled toward the pupil, an adjustable opening whose size is controlled by the iris. The iris, a pigmented muscle, regulates the amount of light entering the eye by contracting or dilating the pupil, thereby ensuring optimal light levels for clear vision.
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Vision01:24

Vision

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Vision is the result of light being detected and transduced into neural signals by the retina of the eye. This information is then further analyzed and interpreted by the brain. First, light enters the front of the eye and is focused by the cornea and lens onto the retina—a thin sheet of neural tissue lining the back of the eye. Because of refraction through the convex lens of the eye, images are projected onto the retina upside-down and reversed.
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Glaucoma: Overview01:25

Glaucoma: Overview

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Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
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Updated: Jan 9, 2026

Development of a Gaze-Contingent Display Framework Designed for Perceptual and Oculomotor Research with Simulated Central Vision Loss
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Development of a Gaze-Contingent Display Framework Designed for Perceptual and Oculomotor Research with Simulated Central Vision Loss

Published on: April 11, 2025

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Functional visual loss.

Shanil Dhanji1, Mitchell Lawlor

  • 1aSave Sight Institute, The University of Sydney bSydney Eye Hospital, Sydney, New South Wales, Australia.

Current Opinion in Neurology
|February 1, 2017
PubMed
Summary
This summary is machine-generated.

Functional visual loss (FVL) is a complex neurological condition where vision tests don't match patient reports. Understanding FVL is advancing, with new insights into its causes and treatments.

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Area of Science:

  • Neurology
  • Ophthalmology
  • Psychiatry

Background:

  • Functional visual loss (FVL) presents a discrepancy between subjective visual experience and objective clinical findings.
  • Diagnosis requires excluding organic causes and confirming normal function via examination or electrophysiology.
  • Recent diagnostic criteria acknowledge that FVL may not always involve a clear psychological association.

Purpose of the Study:

  • To explore recent advancements in the pathophysiology and management of functional disorders, specifically focusing on FVL.
  • To review current understanding and treatment strategies for functional visual loss.

Main Methods:

  • Review of recent literature on functional disorders and FVL.
  • Analysis of diagnostic criteria and neuroimaging findings.
  • Evaluation of treatment evidence for FVL and associated comorbidities.

Main Results:

  • FVL diagnosis necessitates ruling out organic pathology and confirming normal function.
  • Emerging evidence suggests stress-related prefrontal cortex suppression may impact visual awareness.
  • Limited treatment guidelines exist, with education, reassurance, CBT, and pharmacotherapy for comorbidities being primary approaches.

Conclusions:

  • Functional visual loss remains poorly understood, highlighting the need for further research.
  • Advances in understanding functional disorders generally may inform FVL research.
  • Integrated approaches addressing neurological and potential psychiatric aspects are crucial for FVL management.