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A newly identified MHV-A59 ORF1a polypeptide p65 is temperature sensitive in two RNA negative mutants

S A Hughes1, M R Denison, P Bonilla

  • 1University of Pennsylvania School of Medicine, Phila.

Insights

Researchers identified a 65-kDa polypeptide (p65) in MHV-A59 infected cells, crucial for viral life cycle. Its absence in mutants suggests p65 plays a vital role in virus replication.

Area of Science:

  • Virology
  • Molecular Biology
  • Cellular Biology

Background:

  • Murine hepatitis virus (MHV-A59) gene 1 encodes essential viral polypeptides.
  • Understanding the processing and function of these polypeptides is key to deciphering viral replication mechanisms.

Purpose of the Study:

  • To identify and characterize polypeptide products of MHV-A59 gene 1, specifically focusing on a 65-kDa polypeptide (p65) found in infected cells.
  • To investigate the kinetics of p65 appearance and processing in relation to other viral proteins.
  • To compare in vivo and in vitro processing of gene 1 products.

Main Methods:

  • Infected cell lysate analysis using immunoprecipitation.
  • In vitro translation of MHV-A59 genomic RNA.
  • Kinetic studies of polypeptide appearance and processing.
  • Analysis of temperature-sensitive (ts) mutants.

Main Results:

  • A 65-kDa polypeptide (p65) encoded in ORF 1a was identified in MHV-A59 infected DBT cell lysates.
  • p65 appeared after p28 but before larger polypeptides (p290, p240, p50).
  • No p65 homologue was detected in in vitro translations, indicating differences in processing between in vivo and in vitro systems.

Conclusions:

  • In vitro processing of MHV-A59 gene 1 products differs from late-stage intracellular processing.
  • p65 is likely important for the MHV-A59 virus life cycle, as evidenced by its absence in specific ts mutants at non-permissive temperatures.

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