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A low-copy number plasmid mediating beta-lactamase production by Xanthomonas maltophilia

M D Kelly1, J E Mortensen

  • 1Cardeza Foundation for Hematological Research, Thomas Jefferson University, Philadelphia, PA, USA.

Insights

This study investigated antimicrobial resistance in Xanthomonas maltophilia by analyzing plasmid DNA. A specific plasmid, pTHB, was found to confer resistance to penicillins and cefazolin when transferred to E. coli.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Xanthomonas maltophilia frequently exhibits high levels of antimicrobial resistance.
  • Understanding the genetic basis of this resistance is crucial for effective treatment strategies.

Purpose of the Study:

  • To identify and characterize plasmids responsible for antimicrobial resistance in clinical isolates of Xanthomonas maltophilia.
  • To determine the specific resistance mechanisms conferred by these plasmids.

Main Methods:

  • Plasmid DNA was isolated from five clinical isolates of Xanthomonas maltophilia.
  • Plasmid DNA was analyzed, and specific plasmids were purified.
  • Plasmid transformation into E. coli HB101 was performed to assess gene expression.

Main Results:

  • Two plasmids, pXM222 (6.5 kb) and pTHB (5.6 kb), were identified in a single isolate.
  • Transformation of the pTHB plasmid into E. coli HB101 resulted in the expression of resistance.
  • The pTHB plasmid conferred resistance to all tested penicillins and cefazolin.

Conclusions:

  • The pTHB plasmid plays a significant role in conferring penicillin and cefazolin resistance in Xanthomonas maltophilia.
  • Plasmid-mediated resistance mechanisms are a key factor in the antimicrobial resistance of Xanthomonas maltophilia.

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