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Identification and analysis of MHV-A59 P28 cleavage site

S A Hughes1, P J Bonilla, S R Weiss

  • 1Department of Microbiology, University of Pennsylvania School of Medicine, Philadelphia 94104-6076, USA.

Insights

Murine hepatitis virus (MHV-A59) proteolysis of the ORF1a polyprotein involves cleavage of p28. Specific amino acid substitutions at the cleavage site, particularly at P1 and P2, significantly inhibit this process, revealing a novel viral protease recognition sequence.

Area of Science:

  • Virology
  • Molecular Biology
  • Protease Function

Background:

  • Murine hepatitis virus (MHV-A59) is an important model for coronavirus research.
  • The MHV-A59 ORF1a polyprotein undergoes proteolytic processing to generate functional viral proteins.
  • Understanding viral protease specificity is crucial for developing antiviral strategies.

Purpose of the Study:

  • To identify the precise cleavage site of the p28 polypeptide during MHV-A59 ORF1a translation.
  • To investigate the role of specific amino acid residues in the p28 cleavage event.
  • To characterize the recognition sequence of the viral papain-like proteinase.

Main Methods:

  • Amino terminal radiosequencing to determine the exact cleavage point.
  • Site-directed mutagenesis to alter amino acids surrounding the cleavage site.
  • Analysis of proteolysis efficiency following mutagenesis.

Main Results:

  • Cleavage of p28 occurs precisely between Gly247 and Val248.
  • Mutations at Arg246 (P2) and Gly247 (P1) drastically reduced p28 cleavage.
  • Most other amino acid substitutions (P7 to P2') were tolerated, but some reduced proteolysis.

Conclusions:

  • The p28 cleavage site is defined by specific amino acid residues, with P1 and P2 being critical.
  • These findings elucidate a novel recognition sequence for the MHV-A59 papain-like proteinase.
  • This knowledge contributes to understanding coronavirus polyprotein processing and protease mechanisms.

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