Related Experiment Videos

Abnormal gene expression causing hyperammonemia in carnitine-deficient juvenile visceral steatosis (JVS) mice

T Saheki¹, M Tomomura, M Horiuchi

¹Department of Biochemistry, Faculty of Medicine, Kagoshima University, Japan.

Advances in Experimental Medicine and Biology

|January 1, 1997

Abstract

No abstract available in PubMed .

Related Concept Videos

Articles linked to this work by shared authors, journal, and citation graph.

Determination of amino acid sequence responsible for suppression of bone resorption by serum calcium-decreasing factor (caldecrin).

FEBS letters·2001

Role of the conserved Ser-Tyr-Lys triad of the SDR family in sepiapterin reductase.

Chemico-biological interactions·2001

Caldecrin is a novel-type serine protease expressed in pancreas, but its homologue, elastase IV, is an artifact during cloning derived from caldecrin gene.

Journal of biochemistry·1998

Suppressed expression of the urea cycle enzyme genes in the liver of carnitine-deficient juvenile visceral steatosis (JVS) mice in infancy and during starvation in adulthood.

Journal of biochemistry·1997

Long-chain fatty acids suppress the induction of urea cycle enzyme genes by glucocorticoid action.

FEBS letters·1996

Molecular cloning and expression of human caldecrin.

FEBS letters·1996

See all related articles