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American Journal of Physiology. Renal Physiology|June 19, 2015
Distinct α-intercalated cell morphology and its modification by acidosis define regions of the collecting ductJeffrey M Purkerson, Andrew L Schwaderer, Aya Nakamori, et al.American Journal of Physiology. Renal Physiology|September 1, 2005
Galectin-3 expression is induced in renal beta-intercalated cells during metabolic acidosisAndrew L Schwaderer, Soundarapandian Vijayakumar, Qais Al-Awqati, et al.American Journal of Physiology. Renal Physiology|December 17, 2019
Acidosis induces antimicrobial peptide expression and resistance to uropathogenic E. coli infection in kidney collecting duct cells via HIF-1αHu Peng, Jeffrey M Purkerson, Robert S Freeman, et al.Pediatric Nephrology (Berlin, Germany)|July 7, 2023
Application of GFR estimating equations to children with normal, near-normal, or discordant GFRAndrew L Schwaderer, Paula Maier, Larry A Greenbaum, et al.American Journal of Physiology. Renal Physiology|August 22, 2014
Carbonic anhydrase 2 deficiency leads to increased pyelonephritis susceptibilityDavid S Hains, Xi Chen, Vijay Saxena, et al.International Journal of Nephrology|September 20, 2016
Acute Kidney Injury and Atypical Features during Pediatric Poststreptococcal GlomerulonephritisRose M Ayoob, Andrew L SchwadererAnnals of Translational Medicine|February 21, 2017
The association between bacteria and urinary stonesAndrew L Schwaderer, Alan J WolfeAmerican Journal of Physiology. Renal Physiology|May 12, 2020
Developmental loss, but not pharmacological suppression, of renal carbonic anhydrase 2 results in pyelonephritis susceptibilityJohn Ketz, Vijay Saxena, Samuel Arregui, et al.Pathogens (Basel, Switzerland)|February 6, 2016
Genetic Variations in Vesicoureteral Reflux SequelaeDavid S Hains, Andrew L SchwadererUrolithiasis|March 10, 2017
Urinary stone disease in pediatric and adult metabolic bone clinic patientsAndrew L Schwaderer, Abimbola Oduguwa, Kirsten KusumiPageof 19