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Cancer Letters|January 18, 2017
p53 mediates the suppression of cancer cell invasion by inducing LIMA1/EPLINTomoko Ohashi, Masashi Idogawa, Yasushi Sasaki, et al.
Cureus|December 26, 2025
Early Detection of Neural Fatigue in Clinically Healthy Postpartum Mothers Using Functional Near-Infrared SpectroscopyRyohei Kimura, Tomoyuki Ueda, Tomoko Ohashi, et al.
The Journal of Veterinary Medical Science|November 7, 2002
Influence of canine cumulus oophorus on homologous sperm motilityEiichi Kawakami, Tomoko Ohashi, Tatsuya Hori, et al.
Yakugaku Zasshi : Journal of the Pharmaceutical Society of Japan|April 6, 2007
[Visualization and evaluation of the promoter activities of genes for stress-inducible proteins in response to environmental pollutants]Tomoko Ohashi, Tetsuya Satoh, Minoru Kuwata, et al.
International Journal of Cancer|March 16, 2017
Long non-coding RNA NEAT1 is a transcriptional target of p53 and modulates p53-induced transactivation and tumor-suppressor functionMasashi Idogawa, Tomoko Ohashi, Yasushi Sasaki, et al.
Molecular Cancer Research : MCR|October 22, 2013
AKR1B10, a transcriptional target of p53, is downregulated in colorectal cancers associated with poor prognosisTomoko Ohashi, Masashi Idogawa, Yasushi Sasaki, et al.
Oncotarget|October 4, 2014
Array-based genome-wide RNAi screening to identify shRNAs that enhance p53-related apoptosis in human cancer cellsMasashi Idogawa, Tomoko Ohashi, Jun Sugisaka, et al.
FEBS Letters|February 1, 2002
Rapid oxidation of dichlorodihydrofluorescin with heme and hemoproteins: formation of the fluorescein is independent of the generation of reactive oxygen speciesTomoko Ohashi, Atsushi Mizutani, Akira Murakami, et al.
Food and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|May 12, 2018
Effects of high fructose intake on liver injury progression in high fat diet induced fatty liver disease in ovariectomized female miceTomoko Ohashi, Masaki Kato, Akihiro Yamasaki, et al.
Oncogene|December 13, 2019
p53-induced ARVCF modulates the splicing landscape and supports the tumor suppressive function of p53Natsumi Suzuki, Masashi Idogawa, Shoichiro Tange, et al.
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